Dr. Phlip McMillan,  John McMillan

Imagine a masked medical trio in green scrubs under operating lights, digging with their scalpels, carefully extracting strange, rubbery string-like structures from the blood vessels of a cadaver and arranging them on the stainless steel table. This isn’t fiction. It’s a real-world phenomenon being documented by embalmers across the world, unfolding largely unseen by the public and unaddressed by mainstream medical discourse. These professionals are encountering unusual fibrous, rubbery clots, sometimes remarkably long, that bear little resemblance to typical post-mortem blood coagulation. Adding another layer to this medical puzzle, recent scientific analyses suggest some of these formations exhibit fluorescence under specific lighting conditions. What exactly are these fibrous clots? And what might their peculiar glowing properties tell us about their origin?

These formations, often referred to as “white fibrous clots,” aren’t your typical blood clots. They’re described as extraordinarily resilient, dense, and often incredibly long – picture tissue strings stretching up to 19 inches. Dr. Philip McMillan, a researcher and clinician who has been closely following these developments, notes they are “nothing like we have really seen before”. Initially dismissed by some as merely an artifact of death or embalming chemicals, the narrative shifted when insiders, whistleblowers from medical settings, shared evidence of similar startlingly large, white and red clots being extracted from the major veins of living individuals in clinical settings, such as catheterization labs. This raises unsettling questions about their potential presence and impact within the body prior to death. Could these be forming, silently, inside people?

Fluorescent Clots: A Biochemical Mystery

One of the most startling recent findings, brought to light by researcher Kevin McCairn, concerns the clots’ behavior under ultraviolet (UV) light in the 250–500 nm excitation range. McCairn reported a “moderate to green fluorescent intensity” that was strikingly uniform across the entire clot structure. Think about things that glow under a blacklight – usually specific dyes or minerals. While natural fluorescence exists in the body—components like NADH, certain flavins, amino acids (tryptophan), collagen, and even hemoglobin can glow under specific conditions—this uniform, intense emission from the entirety of these large clots is considered highly unusual. It suggests a widespread biochemical modification within the clot’s structure. Researchers are exploring several hypotheses for this glow, pointing towards biochemical changes perhaps related to sugars (Advanced Glycation End products), phosphate groups (hyperphosphorylation), or even the bizarre incorporation of metals like tin, which has reportedly been found in high concentrations within some samples.

Investigating the composition of these clots offers further clues. Lab analyses show significant amounts of fibrin, the protein mesh that forms normal clots. But they also contain high amounts of hemoglobin – the oxygen-carrying molecule usually tucked safely inside red blood cells. Its presence outside cells and integrated into the clot structure is peculiar. Dr. McMillan speculates on a possible connection, noting that the SARS-CoV-2 spike protein is known to bind free hemoglobin, potentially concentrating it within these structures alongside fibrin. Further research by professors Resia Pretorius and Douglas Kell has documented persistent “fibrinoid microclots” in patients with Long COVID. Using fluorescence microscopy, they observed that even these tiny microclots light up when exposed to spike protein, suggesting a potential link between the spike protein, amyloid-like fibrin structures, and fluorescence. This invites the question: are the huge clots seen by embalmers simply scaled-up versions of these troublesome microclots?

It’s a perplexing situation, made more so by the stunning silence from the broader scientific community. Despite observations accumulating from independent researchers, embalmers, and clinicians, there appears to be a curious absence of comprehensive research studies or large-scale investigation. Dr. McMillan voices a sense of obligation: “For me, I have to look at it… there is a scientific responsibility to at least consider and reflect on what is happening.” This apparent institutional inertia feels jarringly familiar to those familiar with medical history.

Historical Parallels: Scientific Resistance to Paradigm Shifts

Consider Ignaz Semmelweis in the 1840s, who observed doctors unwittingly transmitting infection from autopsy rooms to delivery wards, resulting in maternal fatalities. His evidence that handwashing dramatically reduced postpartum infections faced institutional resistance, partly because it challenged established medical thinking and lacked a theoretical framework (as germ theory wasn’t yet established). Similarly, in the 1980s, Barry Marshall and Robin Warren encountered skepticism when proposing that Helicobacter pylori bacteria, not stress, caused stomach ulcers. Their hypothesis eventually earned a Nobel Prize after Marshall’s dramatic self-experimentation provided irrefutable evidence. In both cases, crucial observations challenging the status quo were ignored, costing lives and delaying progress.

Unlike these historical examples, today’s medical establishment has sophisticated imaging technologies and molecular analysis tools to investigate these claims, and our understanding of clotting pathophysiology is far more advanced. The question remains whether these modern advantages will facilitate faster recognition and investigation of this phenomenon, or whether institutional inertia will delay scientific progress as it has in the past.

Potential Mechanisms and the Call for Scientific Investigation

So, what might be the explanation for these fluorescent clots? Several potential mechanisms are being explored, including the role of the SARS-CoV-2 spike protein, whether from infection or potentially persisting after vaccination. Some research points towards specific genetic segments of the virus, like ORF10, potentially making blood “more likely to clot.” Another prominent hypothesis involves an autoimmune reaction. Dr. McMillan suggests the clots might form rapidly due to a “quick autoimmune process,” perhaps analogous to the rare but serious Heparin-Induced Thrombocytopenia (HIT), where the immune system mistakenly attacks platelets, leading to widespread clotting. The rapid onset observed in some reported cases—patients becoming critically ill within days—lends credence to this possibility. Endothelial dysfunction and altered red blood cell behavior are also implicated.

The silence and lack of concerted investigation aren’t just academic concerns. If these clots signify an underlying, widespread pathology linked to recent biological events, understanding them is paramount for public health. Dismissing the evidence without thorough investigation risks eroding trust in scientific institutions, recalling historical failures to address concerns like those surrounding asbestos or thalidomide until harm became undeniable.

What’s needed now is transparency and rigorous science. We need pathologists, biochemists, immunologists, and clinicians working together, examining samples, testing hypotheses, and seeking answers without fear or favor. As Dr. McMillan states simply, reflecting the core of the issue, “something isn’t right.” Will the scientific community rise to the challenge this time, or will it again miss another potentially vital warning from an unexpected source?