Dr. Philp McMillan, John McMillan
The official dashboard tells a reassuring story. According to the UK government’s weekly mortality reports for England and Wales, deaths registered in mid-November ran 4.5% below expected levels. The black trend line on the chart, which represents the threshold for concern, remains safely above the actual figures. Case closed. Move along.
But pull the raw registration data from 2015 to 2024 and plot it yourself, and a different picture emerges. It is one that statistical modeling has quietly obscured. Following any mass casualty event, epidemiologists expect what they call “mortality debt,” a compensatory drop in deaths below baseline. The logic is grimly straightforward: people can only die once. When a pandemic sweeps through a population and claims the vulnerable: the elderly, the immunocompromised, and those with kidney disease, heart failure, and dementia, those individuals are removed from future mortality statistics. The death rate should, mathematically, fall below historical trends for a period afterward. This has not happened.
When England and Wales data from 2022 through 2024 is compared against the 2015-2019 trend line, deaths remain stubbornly elevated. Not by a small margin. The numbers sit well above where they should be, and crucially, well above where they would sit if the expected mortality debt had been paid. The gap represents tens of thousands of unexplained deaths. The Australian parliament acknowledged excess deaths and chose not to investigate. The UK position has been simpler: deny the excess exists at all. The statistical models were adjusted, the baseline recalculated, and the inconvenient reality was smoothed away.
What the Autopsies Revealed
The numbers demand an explanation. And when statistics fail to provide one, pathology must. A German autopsy study published in November 2022 examined 25 individuals who died unexpectedly within 20 days of COVID vaccination. The clinical context matters enormously: these were not people who felt ill enough to seek medical attention. They went to bed and did not wake up. They collapsed without warning. The mean time from vaccination to death was two and a half days. Of the 25 cases examined, five (a full 20%) showed acute myocarditis. Heart inflammation. In the absence of competing explanations, this inflammation stands as the most plausible mechanism of death. The histopathology tells a specific story. And it is not a story of ordinary inflammation.
The Missing Brake Cells
When pathologists examined the cardiac tissue, they found something that should have triggered alarm bells across the immunology community. The inflammatory infiltrates, or clusters of immune cells concentrated in patches throughout the heart muscle, were missing a critical component. FOXP3-positive regulatory T cells were absent. These cells function as the immune system’s brakes. When inflammation occurs anywhere in the body, regulatory T cells move into the area to moderate the response and prevent the immune system from overshooting its target and damaging healthy tissue. Their absence in these cardiac infiltrates suggests an immune response running without restraint.
Protected Cells, Collateral Damage
But the more striking finding involves a cellular protection mechanism that appears to be driving the pathology. When lipid nanoparticles deliver modified mRNA into a muscle cell, including cardiac muscle, that cell begins producing spike protein on its surface. Under normal circumstances, this should mark the cell for immune destruction. The immune system should recognize the foreign protein and eliminate the cell producing it. Instead, something else happens. The spike-producing cells upregulate PD-L1, a protein that effectively shields them from immune attack. It is as if the cell has posted a legal notice on its door: the police gather outside, warrant in hand, but cannot enter. The immune cells do not simply disperse. They remain clustered around the protected cell, frustrated and activated, and they begin attacking the surrounding tissue instead. Healthy cardiac muscle cells—meaning cells not producing spike protein and cells with no legal protection—are destroyed in the crossfire. This creates focal zones of inflammation and micro-scarring scattered throughout the heart. These micro-scars become the substrate for fatal arrhythmias. The heart’s electrical conduction system depends on the uniform transmission of electrical signals across healthy tissue. Introduce patches of scar tissue, and those electrical signals fragment. They circle back on themselves. The coordinated muscle contraction that keeps blood moving becomes chaotic and ineffective. The heart stops pumping blood efficiently.
The timeline matters here. Two and a half days is too short for this entire process to develop from scratch. The pathology suggests these inflammatory patterns were already established, likely from earlier vaccine doses or prior infection, and were reactivated upon subsequent spike protein exposure. Each dose potentially adds new foci of damage. Each exposure compounds the risk.
When Priming Meets Re-Exposure
The persistent excess deaths and the autopsy findings are not separate phenomena requiring separate explanations. They are cause and effect. Dr. Philip McMillan, who has tracked these patterns since 2021, frames the mechanism in memorable terms. Vaccination creates immune priming, where the system learns to recognize and react to spike protein. Subsequent COVID infection delivers that same spike protein again. “On their own, they don’t really stick,” he observes of these two factors. “But if you put them together, you then have an absolutely frightening situation.” This is not a temporary risk that fades with time. As long as COVID continues circulating, and it shows no signs of disappearing, the vaccinated (and some unvaccinated) population faces repeated re-exposure to the protein their immune systems have been trained to attack. Each encounter carries the potential to reactivate the inflammatory patterns, to add new damage to old, and to push a heart already scarred closer to electrical failure. The mortality debt will not be paid because the conditions creating excess deaths have not resolved. They have become endemic.
The Bodies Remain
Statistical manipulation cannot alter biological reality. The dashboards can be adjusted, the baselines recalculated, and the excess defined out of existence. But the bodies remain. “Without autopsies, we are flying blind,” Dr. McMillan warns. The German study represents one of the few systematic attempts to examine what is actually happening at the tissue level in post-vaccination deaths. The findings are significant. The absence of follow-up research is more significant still. The path forward requires acknowledging what the raw data already shows: something continues to kill people at rates above historical norms. The autopsy evidence points toward a specific mechanism, which is immune dysregulation triggered by spike protein exposure in primed individuals. Until this is investigated with the seriousness it demands, the excess deaths will persist. And the debt will remain unpaid.
References:
Deaths registered weekly in England and Wales, provisional
0 Comments